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Barnes, Kimberly

Interim Associate Dean of Academic Affairs
Associate Professor of Biochemistry

Dr. Barnes serves as coordinator of the Intercollegiate Biochemistry Program. She earned a bachelor of science degree from Michigan State University in 2000, a master of science degree from the University of Nebraska in 2002, and a doctorate from the University of Nebraska in 2005, all in animal science. She served as a Postdoctoral Fellow in the Department of Nutritional Sciences at the University of Wisconsin-Madison (2005 – 2007) and then as an Assistant – Associate Professor of Biochemistry in the Division of Animal and Nutritional Sciences at West Virginia University (2007 – present).  In 2013 the Intercollegiate Biochemistry Program was formed and Dr. Barnes took over the coordinator role in 2015.

Her research focused on dietary fatty acids and their role in lipid metabolism and adipose tissue biology.  Specifically her laboratory focused on dietary Conjugated Linoleic Acid (CLA) and its interaction with other dietary fatty acids.  A main goal of this work was to determine the molecular mechanism by which CLA causes an increase in lipolysis, and greater loss of body fat, in mice fed coconut oil than in mice fed soy oil.  An additional project focused on different sources of omega-3 fatty acids and their effectiveness at reducing serum lipids.  Specifically omega-3 fatty acids from algal and yeast sources were less effective at being incorporated into tissues and at effecting serum triglyceride and cholesterol levels than those coming from fish oil.

Select Publications

  1. Ippagunta, S, Z Angius, M Sanda, KM Hargrave-Barnes.  2013.  Dietary CLA-induced lipolysis is delayed in soy oil-fed mice compared to coconut oil-fed mice.  Lipids 48(11):1145-1155.
  2. Kanosky, KM, S Ippagunta, and KM Barnes.  2013.  Mice do not accumulate muscle lipid in response to dietary conjugated linoleic acid.  J. Anim. Sci. 91:4705-4712.
  3. Shelton, VJ, AG Shelton, MJ Azain, and KM Hargrave-Barnes.  2012.  Incorporation of conjugated linoleic acid (CLA) into brain lipids is not necessary for CLA-induced reductions in feed intake or body fat in mice.  Nutr. Res. 32:827-836.
  4. Barnes, KM, NR Winslow, AG Shelton, KC Hlusko, and MJ Azain.  2012.  Effect of dietary conjugated linoleic acid on marbling and intramuscular adipocytes in pork.  J. Anim. Sci. 90:1142-1149.
  5. Ippagunta, S, T.J. Hadenfeldt, JL Miner, and KM Hargrave-Barnes.  2011.  Dietary conjugated linoleic acid induces lipolysis in adipose tissue of coconut oil-fed mice but not soy oil-fed mice.  Lipids 46:821-830.
  6. Hausman, GL, MV Dodson, K Ajuwon, M Azain, KM Barnes, LL Guan, Z Jiang, SP Poulos, RD Sainz, S Smith, M Spurlock, J Novakofski, ME Fernyhough, and WG Bergen. 2009.  Board Invited Review: The biology and regulation of preadipocytes and adipocytes in meat animals. J. Anim. Sci. 87:1218-1246.
  7. Hargrave-Barnes, KM, MJ Azain, and JL Miner.  2008. Conjugated linoleic acid-induced fat loss dependence on 6-desaturase or cyclooxygenase.  Obesity 16:2245-2252.
  8. Hargrave, KM, and JL Miner. 2006. The trans-10,cis-12 CLA isomer induces death of 3T3-L1 preadipocytes but not adipocytes. Adipocytes 2:125-132.
  9. Hargrave, KM, MJ Azain, and JL Miner. 2005. Dietary coconut oil increases the sensitivity to conjugated linoleic acid-induced body fat loss in mice independent of an essential fatty acid deficiency. Biochim. Biophys. Acta 1737:52-60.
  10. Hargrave, KM, BJ Meyer, CL Li, MJ Azain, CA Baile, and JL Miner. 2004. Influence of conjugated linoleic acid and fat source on body fat and apoptosis in mice. Obes. Res. 12:1435-1444.
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